Philip M. Parker's Amyotrophic Lateral Sclerosis - A Bibliography and PDF

By Philip M. Parker

ISBN-10: 0497113279

ISBN-13: 9780497113278

ISBN-10: 142949610X

ISBN-13: 9781429496100

In March 2001, the nationwide Institutes of wellbeing and fitness issued the subsequent caution: ''The variety of sites supplying health-related assets grows each day. Many websites supply priceless details, whereas others could have info that's unreliable or misleading.'' moreover, a result of swift elevate in Internet-based details, many hours might be wasted looking out, choosing, and printing. on the grounds that merely the smallest fraction of knowledge facing Amyotrophic lateral sclerosis is listed in se's, comparable to www.google.com or others, a non-systematic method of net examine should be not just time eating, but additionally incomplete. This booklet was once created for doctors, scholars, and participants of most of the people who are looking to behavior scientific learn utilizing the main complex instruments to be had and spending the smallest amount of time doing so.

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Additional info for Amyotrophic Lateral Sclerosis - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers

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24 Amyotrophic Lateral Sclerosis measurements in normal mice with partially inhibited electron transport chain activity, one of the proposed mechanisms for ALS pathogenesis. Young mice that have yet to develop signs of disease will be used and compared to previous results from older, affected mice. Finally, the degree of denervation of motor end-plates after marked neuronal activity will be measured with fluorescent markers for both the nerve terminal cytoplasm and the muscle end-plate. ; Massachusetts General Hospital 55 Fruit St Boston, Ma 02114 Timing: Fiscal Year 2004; Project Start 01-AUG-2001; Project End 31-JUL-2006 Summary: (provided by applicant): The goal of this project is to investigate the role of a newly discovered postsynaptic protein, Shank, in the regulation of dendritic spine morphology and cytoskeleton.

The finding that the genes encoding these proteins are mutated in some familial forms of these diseases strongly argues that these aggregating proteins cause these diseases. However, for all these diseases the relationship between protein aggregation and cellular pathology has not been clearly established. It is also unknown if the common association of protein aggregation with these diseases reflects a common underlying toxic mechanism, or, alternatively, a common downstream result of cell pathology.

Secondary endpoints include the rate of decline of forced vital capacity (FVC, percent predicted), the change in the ALS functional rating scale (ALSFRS-R), the SF-12 (quality of life instrument), and the safety and tolerability of creatine. ; Professor & Vice Chairman; Neurology; University of Pittsburgh at Pittsburgh 350 Thackeray Hall Pittsburgh, Pa 15260 Timing: Fiscal Year 2004; Project Start 01-JUN-1999; Project End 31-MAY-2008 Summary: (provided by applicant): Cyclooxygenase, the enzyme that catalyzes the production of prostaglandins from arachidonic acid, has long been thought to play a 36 Amyotrophic Lateral Sclerosis role in exacerbating injury due to cerebral ischemia via its vascular and inflammatory effects.

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Amyotrophic Lateral Sclerosis - A Bibliography and Dictionary for Physicians, Patients, and Genome Researchers by Philip M. Parker


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